Hemoconcentration causes a slowing down of the circulatory system, favoring the formation of thrombi. However, the hematocrit is elevated, that is, there is hemoconcentration due to platelet accumulation and fluid loss. On the other hand, there is massive destruction of red blood cells with the appearance of anemia.
All of these fluid depletion events can cause hypovolemic shock.
CAUSES OF LOW HEMATOCRIT AND HEMOGLOBIN SERIES
Hyperkalemia triggers a series of signs and symptoms in the patient, such as: fatigue, decreased muscle tone, cardiac arrest, paralytic ileus, among others.
Loss of fluid, both through evaporation and edema, causes an electrolyte imbalance at the plasma level characterized by a decrease in sodium (hyponatremia) and an increase in potassium (hyperkalemia).
CAUSES OF LOW HEMATOCRIT AND HEMOGLOBIN SKIN
Through this route, up to 7 liters per day can be lost in patients with a large area of affected skin (≥ 50%). Burned skin is unable to retain moisture and, on the contrary, gives off water vapor. In addition, in the burned patient there is loss of liquid by massive evaporation. All the above mentioned contributes to the formation of a large interstitial edema. Likewise, there is less venous reabsorption due to the decrease in oncotic pressure. Subsequently, the high concentration of proteins accumulated in the interstitial fluid further favors the attraction of water. This causes the albumin to move into the interstitial space. When the skin burns, there is an alteration in capillary permeability due to the increase in the concentration of histamine. In the burned patient, a series of events occur that clarify why hemoconcentration occurs and how hypovolemic shock can occur. Hemoconcentration and blood loss can lead to hypovolemic shock that can lead to death. On the other hand, the virus causes the destruction of multiple types of cells, including T lymphocytes and platelets, which translates into a decrease in the patient's immunity and the appearance of significant bleeding. The disease is characterized by an increase in the secretion of cytokines that favors the extravasation of the plasma to the extravascular space, producing hemoconcentration. These antibodies favor the replication of the virus and the increase in viremia in the second infection, causing a serious picture of the disease called hemorrhagic dengue. The first infection leaves heterologous antibodies. The severe form of the disease occurs when there is reinfection by another serotype other than the first. The virus enters the patient by the bite of a blood-sucking vector called Aedes aegypti. Hemoconcentration in dengueĭengue is a viral infection caused by an arbovirus of the Flaviviridae family. The loss of fluid causes a decrease in plasma volume and the consequent hemoconcentration. In the neonatal period, there may be physiological hemoconcentration, but after this period, such a high level of hemoglobin (> 20 g / dl) is alarming and dangerous. Hemoconcentrated patients usually have hemoglobins above 17 g / dl. It occurs in dehydrated patients, in large burns, in dengue hemorrhagic fever or in patients with systemic capillary leak syndrome. The imbalance causes plasma extravasation to the extravascular or interstitial space. Hemoconcentration occurs in the event of fluid loss or due to an imbalance in their distribution within the body. That is, although there is an increase in the hematocrit, the amount of red blood cells does not change. The hemoconcentration is the increase in hematocrit concentration in response to the decrease in plasma volume. Differential diagnosis between hemoconcentration and polycythemia.Hemoconcentration in patients with systemic capillary leak syndrome.Hemoconcentration in patients with heart failure.Hemoconcentration in dehydrated patients.Hemoconcentration: causes, consequences and diagnosis - science Content: Hemoconcentration: causes, consequences and diagnosis
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